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Condition-specific correlation between gene expression of inflammatory molecules and colorectal sensitivity. Colorectal sensitivity was positively correlated with TGF-β2 (A) in control, and with IL-1β (B), inhibin βA (C) and PGES (D) in Dex-treated post-TNBS mice. Broken lines in B-D indicate statistically insignificant linear regressions in Veh-treated post-TNBS mice. (E) Gene expression of COX-2 was negatively correlated with colorectal sensitivity in AHS-treated mice.
