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. 2014 Nov 28;5:600. doi: 10.3389/fimmu.2014.00600

Table 4.

KIR3DS05, KIR3DL10 epistasis with MHC class I alleles on HVL risk.

KIR3DS05 KIR3DL10

MHC class I
KIR3DS05
Epistasis
Pa MHC class I
KIR3DL10
Epistasis
Pa
MHC class I ORMHC (p value) ORKIR (p value) ORMHCxKIR (p value) ORMHC (p value) ORKIR (p value) ORMHCxKIR (p value)
A1*001 1.50 (p = 0.622) 21.60 (p = 0.009) 0.035 (p = 0.020) 0.041 1.40 (p = 0.654) 21.60 (p = 0.009) 0.036 (p = 0.024) 0.041
A1*004 11.00 (p = 0.015) 7.33 (p = 0.034) 0.20 (p = 0.224) 3.64 (p = 0.101) 3.25 (p = 0.167) 1.10 (p = 0.942)
B*012 0.50 (p = 0.471) 1.25 (p = 0.755) 10.00 (p = 0.087) 0.40 (p = 0.320) 1.20 (p = 0.811) 25.00 (p = 0.036)
B*046 2.67 (p = 0.283) 4.40 (p = 0.044) 0.26 (p = 0.272) 1.30 (p = 0.745) 3.66 (p = 0.084) 1.03 (p = 0.984)
B*057 6.42 (p = 0.040) 6.60 (p = 0.028) 0.17 (p = 0.153) 2.60 (p = 0.221) 4.33 (p = 0.089) 0.58 (p = 0.659)
B*072 10.29 (p = 0.047) 20.01 (p = 0.014) 0.054 (p = 0.043) 3.00 (p = 0.179) 8.00 (p = 0.029) 0.29 (p = 0.330)

Displayed are all model term estimates (main effects and epistasis: odds ratio’s OR (p values), p ≤ 0.05 in bold) for twelve models of KIRMHC class I polymorphism pairings. The two correlated KIR risk genes KIR3DS05 and KIR3DL10 were coded for transcript presence at pre-infection. The MHC class I alleles were coded for transcript presence, did not correlate significantly, and were frequent with sufficient strata sizes. The simultaneous risk estimates ORMHC, ORKIR, and ORMHCxKIR quantify HVL risk of MHC-I in KIR-negative animals (ORMHC), HVL risk of KIR in MHC-I-negative animals (ORKIR) and epistatic effects for coincident presence of KIR and MHC-I (ORMHCxKIR). Animals with coincident MHC-I and KIR presence have the HVL risk ORMHC⋅ORKIR⋅ORMHCxKIR compared to animals with coincident MHC-I and KIR absence.

aProbability that – under the null-hypothesis of no epistasis and no genetic associations – two or more p ≤ 0.05 for six epistasis tests of MHC class I alleles with a KIR gene. This probability was estimated based on 10,000 permutations of the HVL/LVL group assignment.