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. 2014 Nov 11;9(1):471–477. doi: 10.3892/ol.2014.2701

Figure 2.

Figure 2

Expression levels of (A) miR-27a and (B) KRAS in various ESCC cell lines. The expression level of each gene is the fold-change relative to the expression level of HEEC (the control). Expression levels of (C) miR-27a and (D) KRAS in 30 lung cancer patient tissues. U6 small nuclear and 18S RNA served as the internal controls in A and C, and B and D, respectively. Data was obtained by reverse transcription-quantitative polymerase chain reaction and each assay was performed in triplicate. *P<0.05; **P<0.01; ***P<0.001. miR, microRNA; KRAS, Kirsten rat sarcoma viral oncogene homolog; ESCC, esophagus squamous cell carcinoma; HEEC, human epithelial esophageal cells.