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. 2014 Dec;34(24):4379–4388. doi: 10.1128/MCB.00740-14

FIG 2.

FIG 2

The interaction between 14-3-3ζ and Atg9A is increased in hypoxia. (A) HA-Atg9A was overexpressed in HEK-293 cells, and cells were treated with normoxia or hypoxia for 12 h. HA-Atg9A was immunoprecipitated and immunoblotted for endogenous 14-3-3ζ. (B) HA–14-3-3ζ was overexpressed in HEK-293 cells, and cells were treated with normoxia or hypoxia for the indicated time periods (M, mock). HA–14-3-3ζ was immunoprecipitated and immunoblotted for endogenous Atg9A.