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. Author manuscript; available in PMC: 2015 Nov 26.

Published in final edited form as: Virus Res. 2014 Jun 7;193:39–51. doi: 10.1016/j.virusres.2014.06.002

Fig. 4 — (A) Stretch (solid line) and release (dotted line) curves for dsDNA in the absence (black) and presence (red) of 10 nM WT HIV-1 NC. A blue dashed line illustrates how the slope (SL) is calculated form the stretching curve, while the area covered by thin gray lines is the hysteresis area. The overstretching midpoint force, which shows how NC alters DNA stability, is also shown. (B) DNA stretch (solid line) and release (dotted line) curves in the absence of protein (black) and in the presence of 5 nM (red) and 20 nM (blue) WT FIV NC. (C) Slope of the transition of WT FIV NC (black) and WT HIV-1 NC (blue). (D) Hysteresis versus concentration of WT FIV NC (black) and WT HIV-1 NC (blue).

Fig. 4 — (A) Stretch (solid line) and release (dotted line) curves for dsDNA in the absence (black) and presence (red) of 10 nM WT HIV-1 NC. A blue dashed line illustrates how the slope (SL) is calculated form the stretching curve, while the area covered by thin gray lines is the hysteresis area. The overstretching midpoint force, which shows how NC alters DNA stability, is also shown. (B) DNA stretch (solid line) and release (dotted line) curves in the absence of protein (black) and in the presence of 5 nM (red) and 20 nM (blue) WT FIV NC. (C) Slope of the transition of WT FIV NC (black) and WT HIV-1 NC (blue). (D) Hysteresis versus concentration of WT FIV NC (black) and WT HIV-1 NC (blue).