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. 2014 Nov 23;9:53. doi: 10.1186/1750-1326-9-53

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© Brinkmalm et al.; licensee BioMed Central Ltd. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Targeted HR-SIM-MS analyses of SNAP-25 in human CSF. Individual values for the HR-SIM-MS measured peak area ratios ([endogenous peptide/labeled peptide standard] multiplied by 10,000) of immunoprecipitated (SMI81 antibody) SNAP-25 in CSF samples within German cohort (A-C), Swedish cohort I (D-F), Swedish cohort II (G-I), and the entire group of Alzheimer’s disease (N = 36) and controls (N = 32) (J-L). The nine panels depict the measured levels of three N-terminal tryptic peptides of SNAP-25, Ac-2-16 (A, D, G, J), 17-31 (B, E, H, K), and 32-40 (C, F, I, L).