|
Wang et al. (
21
)
|
Essential reduction in IL-8 and endotelin-1 (ET-1) levels and no alteration in TNF-a level after MUF. |
|
Bando et al. (
5
)
|
ET-1 levels were extensively lesser after MUF. |
|
Pearl et al. (
22
)
|
MUF does not cause a significant change on thromboxane-B2, ETn-1 and leukotriene-B4 levels post-CPB. |
|
Portela et al. (
23
)
|
Essential reduction in levels of IL-6, intercellular adhesion molecule-1and vascular cell adhesion molecule-1 following MUF. |
|
Yndgaard et al. (
24
)
|
MUF lower circulatory endotoxins in circulation and recurrence of major amounts of this endotoxin load in the ultrafiltration. |
|
Chew et al. (
25
)
|
No intergroup variation visible for TNF-a, IL-1ß and IL-1ra, complements (C3d and C4d). |
|
Huang et al. (
19
)
|
Serum IL-6 levels were considerably lower after MUF, mild thromboxane-B2 was removed and ET-1 levels remained unchanged. |
|
Harig et al. (
26
)
|
MUF led to lesser platelet activation, monocyte shell markers CD45 and CD14 showed clear generations. |
|
Yokoyama et al. (
27
)
|
Removal of prostaglandin E-2 was one cause of augmented blood pressure. |
|
Atkins et al. (
12
)
|
MUF was associated with raised alveolar concentrations of proinflammatory cytokines (IL-6 and IL-8) and trans-pulmonary thromboxane meditations. |
|
Papadopoulos et al. (
28
)
|
MUF was associated with decrease of inflammatory factors, endotoxins and complements. |
