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. 2014 Dec 4;5:419. doi: 10.3389/fgene.2014.00419

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Copyright © 2014 Hermann, Watson and Wildering.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Phospholipase A₂ (PLA₂) contributes to regulation of respiratory neural circuit activity in old and oxidative stressed young brains. Examples of action potential activity recorded from RPeD1 in isolated young central nervous systems (CNSs) pretreated for 30 min with either vehicle-only (top row) or the pro-oxidant 2,2-azobis (2-methylpropion-amidine) dihydrochloride (AAPH; middle row), and old CNSs maintained in vehicle-only (bottom row), before (left column), and after 30 min of treatment with the PLA₂ inhibitor aristolochic acid (right column). Reprinted from Watson et al. (2013) with permission from Elsevier.