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. 2014 Nov 24;15(1):1017. doi: 10.1186/1471-2164-15-1017

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© Boon et al.; licensee BioMed Central Ltd. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Effect of Slc25a13 gene expression on H5N1 influenza virus pathogenesis. (A) Slc25a13-deficient mice (Slc25a13 ^-/-) and control animals were inoculated with 10⁵ HK213 virus for 48 hours and the amount of CCL2 produced in the lungs was measured by ELISA. The results are from 5 mice per group and are representative of two experiments. (B) Slc25a13 ^-/- mice (circles, n =7) and control animals (triangles, n =9) were infected with 10⁵ (filled symbols) or 10⁶ (open symbols) EID₅₀ of a highly pathogenic H5N1 influenza A virus (A/Hong Kong/213/03 (HK213)) and weight-loss was monitored for 15 days.