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. 2014 Mar;59:85–96. doi: 10.1016/j.mcn.2014.02.001

Fig. 7.

Fig. 7

Reduced cytosolic calcium in nAChR mutants may explain PVD defects. A) Basal calcium levels in wild-type, deg-3(u701), des-2deg-3(u773) and cha-1(p1152) animals n = 117, 76, 108, 27 respectively. Differences between mutants and wild-type are significant p < 0.01, one-way ANOVA. B) MCA-3 overexpression leads to a harsh touch defect. Differences between the mec-4(d); MCA-3 overexpressing strain and either mec-4(d) or wild-type (N2) are significant, N = 10–12, n = 10, p < 0.01, one-way ANOVA. C) Above MCA-3 overexpression leads to defects in PVD morphology. Below, distribution of the functional tdtomato::MCA-3a protein in the same cell. Shown are images of late L4-young adults, scale bar 5 μm.