TABLE 2:
Gene | If/Ip ratio ± SEM (n) | p |
---|---|---|
14-3-3hp | 1.51 ± 0.086 (37) | 0.0001 |
rcdBB | 1.64 ± 0.11 (48) | 0.0001 |
mmsdh | 1.46 ± 0.097 (52) | 0.0011 |
rmd1 | 1.37 ± 0.051 (54) | 0.0012 |
DDB_G0272460 | 1.38 ± 0.069 (54) | 0.0032 |
actin | 1.41 ± 0.12 (41) | 0.011 |
corA | 1.31 ± 0.074 (55) | 0.056 |
efbA | 1.31 ± 0.086 (44) | 0.077 |
ctxB | 1.28 ± 0.058 (53) | 0.099 |
14-3-3 | 1.24 ± 0.073 (48) | 0.32 |
ctxA | 1.23 ± 0.058 (34) | 0.34 |
Calcineurin-like | 1.22 ± 0.054 (57) | 0.40 |
TRE5-A ORF | 1.20 ± 0.060 (52) | 0.69 |
rps2 | 1.19 ± 0.061 (48) | 0.73 |
ancA | 1.18 ± 0.053 (50) | 0.90 |
pLD1 vector controla | 1.17 ± 0.034 (91) | |
WT GFP myosin IIb | 2.1 ± 0.2 (29) | 0.0001 |
The background strain was the myoII:3xAsp cells (myoII::pDRH:GFP-3xAsp). n is the number of cells analyzed.
apLD1 vector control was the mutant transformed with the empty vector.
bData for late-stage furrows from Figure 6B.