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. 2014 Jan 1;522(3):592–608. doi: 10.1002/cne.23434

Figure 2.

Figure 2

Confocal images of a female Ae. aegypti AL labeled with antisera against AST-A (green) and synapsin (magenta). All frontal views. A: Maximum projection of 27 optical sections showing the middle to posterior portion of the ALs and the anterior part of the subesophageal ganglion (SOG). Two large cell bodies (arrow) in the SOG send their neurites into the ipsilateral AL, which give rise to thick fibers and varicosities (small arrows) in the center neuropil of the AL. From there, a sparse fiber network invests the glomerular neuropil. Note that the thick fibers never enter the glomeruli but stay outside between them (see also D,G). The neurites extending from the cell bodies indicated by asterisks bypass the ALs. B–D: Maximum projections of two optical sections in the anterior portion of the AL. A group of 12 to 16 cell bodies in the lateral cell group (asterisk) project their neurites into the AL neuropil. The ventral and anteromedial glomeruli show innervations with immunopositive fibers, whereas other glomeruli show little or no immunostaining. Arrows label thick AST-A-immunoreactive varicosities stemming from the posterior meshwork. E–G: A single optical section through the anterior part of the AL shows the ventral and anteromedial glomeruli with AST-A immunostaining. The arrow marks a thick fiber running between two glomeruli. AMMC: antennal motor and mechanosensory center. Scale bars = 20 μm.