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. 2014 Dec 12;34(6):e00160. doi: 10.1042/BSR20140115

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© 2014 The Author(s) This is an Open Access article distributed under the terms of the Creative Commons Attribution Licence (CC-BY) (http://creativecommons.org/licenses/by/3.0/) which permits unrestricted use, distribution and reproduction in any medium, provided the original work is properly cited.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Licence (CC-BY) (http://creativecommons.org/licenses/by/3.0/) which permits unrestricted use, distribution and reproduction in any medium, provided the original work is properly cited.

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Silencing of ANGPTL3 enhances GLUT2-mediated glucose uptake, increases TAG synthesis and induces down-regulation of PGC1α and its downstream targets, including TRB3, hypothetically leading to enhanced Akt phosphorylation levels. Secretion of TAG-enriched VLDL-1-type particles is decreased upon insulin stimulus.