Skip to main content
. 2014 Nov 13;11(3):1615–1622. doi: 10.3892/mmr.2014.2934

Figure 5.

Figure 5

Effect of XNJ on the expression of p-AKT, p-ERK and p-JNK in the striatum following sevoflurane exposure. Upper panel, representative western blots; lower panel, quantitative data showing expression of (A) p-AKT (Ser473), (B) p-AKT (Thr308), (C) p-ERK and (D) p-JNK in the striatum of seven-day-old rats in: Lane 1, the control group; lane 2, the XNJ group; lane 3, the sevo group; lane 4, the sevo plus pre-treatment with 10 ml/kg XNJ group; and lane 5, the sevo plus pre-treatment with 1 ml/kg XNJ group. β-actin was used as a protein loading control. Quantitative data are presented as the mean ± standard deviation (n=5). *P<0.05. XNJ, xingnaojing; Sevo, sevoflurane; p-AKT, phosphorylated protein kinase B; p-ERK, phosphorylated extracellular-regulated kinase; p-JNK, phosphorylated c-Jun N-terminal kinase.

HHS Vulnerability Disclosure