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. Author manuscript; available in PMC: 2016 Jan 1.
Published in final edited form as: Arterioscler Thromb Vasc Biol. 2014 Nov 13;35(1):79–86. doi: 10.1161/ATVBAHA.114.304292

Figure 1.

Figure 1

A: Cathepsin family members were differently expressed in human ruptured carotid endarterectomies, as determined by microarray analysis. Fold changes stable versus ruptured lesion, all p<0.0001. B: Immunohistochemical staining of human carotid atherosclerotic lesions using CatC and CD68 antibodies. Panel (i): representing CatC expression in an early lesion, panel (ii): CatC expression in a stable lesion, panel (iii): CatC expression in a ruptured lesion, panel (iv): parallel section of panel (iii) showing that CatC expression localizes to the same areas of intense CD68-positive staining cells.