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. 2014 Dec 8;3(12):e132. doi: 10.1038/oncsis.2014.48

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Copyright © 2014 Macmillan Publishers Limited

Oncogenesis is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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AKT activation at 0, 14 and 31 days after long-term FR. (a) AKT phosphorylation on Ser473 (P-AKTSer473, active AKT) by FR exposure in AT5BIVA and AT5BIVA/ATM-wt cells (upper panel) and in GM7166 and GM7166/NBS1-wt cells (lower panel). (b) PP2A activity of un-irradiated control and 31 days of FR (0.05 Gy/fraction) in AT5BIVA and AT5BIVA/ATM-wt cells. Cell lysates were collected at 24 h after the last FR. Asterisks indicate a significant decrease in PP2A activity compared with un-irradiated control cells.