Skip to main content
. Author manuscript; available in PMC: 2016 Jan 31.
Published in final edited form as: Biochim Biophys Acta. 2014 Jul 2;1852(2):232–242. doi: 10.1016/j.bbadis.2014.06.030

Figure 1. Interplay of hyperglycemia and peripheral metabolism in cardiometabolic syndrome in mediating diabetic cardiovascular complications.

Figure 1

Hyperglycemia may indirectly (via its metabolic consequences) or directly enhance diabetes-associated inflammation and ROS generation, promoting tissue injury and the development of diabetic cardiovascular and other complications. AT II rec, angiotensin II receptor type 1; CNS, central nervous system; PMNs, polymorphonuclear leukocytes; XO, xanthine oxidase.