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. 2015 Jan;21(1):62–68. doi: 10.5056/jnm14093

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© 2015 The Korean Society of Neurogastroenterology and Motility

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Effect of intracellular Ca²⁺ release or protein kinase A (PKA) inhibition on resveratrol (3,5,4′-trihydroxystilbene, RSVL)-induced relaxation. When the strips were treated with 2-aminoethoxydiphenylborane (2-APB), there was a significant (^*P < 0.05) decrease in the amount of relaxation observed between the strips not treated and treated with 2-APB (n = 6). When the PKA inhibitor 14–22 amide myristolated (PKA-IM), was used, no significant effect was observed on the amount of cholecystokinin octapeptide (CCK)-induced tension; nor was there a significant effect on the amount of RSVL-induced relaxation (n = 6).