Figure 9. In CsA-treated animals, transfusion-associated ABMR correlated with greater inflammation and M1 macrophage phenotype.
Transfusion-induced ABMR in rats was associated with inflammation (iNOS, Nox2) and M1 (iNOS, IRF5) macrophage phenotype. Complement regulatory proteins (Crry, DAF) were not significantly affected by transfusion. ABMR, antibody-mediated rejection; CsA, cyclosporine A; iNOS, inducible nitrogen oxide synthase; IRF5, interferon regulatory factor 5; Nox2, NADPH oxidase 2.