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. 2014 Dec 16;89(1):523–534. doi: 10.1128/JVI.02426-14

TABLE 1.

Summary of HCMV BACs used in this study

Construct Genetic background Reference for background construct Change(s) introduced
WT LMN-ΔH/C WT_E7 AD169-RV Kamil et al. (16) Sequence encoding the dominant negative GFP-lamin A mutant, lacking the lamin A head and CaaX domains, cloned downstream of a duplicated UL97 promoter region in the US9-US10 intergenic region
Δ97 LMN-ΔH/C AD169-RV Hobom et al. (32) Sequence encoding the dominant negative GFP-lamin A mutant, lacking the lamin A head and CaaX domains, cloned to replace the UL97-encoding sequence.
S216A 53-F AD169-RV Sharma et al. (8) Alanine replacement of UL50 serine-216 in UL53-FLAG genetic background
S19A 53-F AD169-RV Sharma et al. (8) Alanine replacement of UL53 serine-19 in UL53-FLAG genetic background
S216A/S19A (AA) 53-F AD169-RV Sharma et al. (8) Double alanine mutant carrying alanine replacements of UL50 serine-216 and UL53 serine-19 in UL53-FLAG genetic background
S216AR/S19AR (RR) AA-53-F AD169-RV This study Rescued derivative of alanine double mutant described above, restoring WT serine-encoding sequences to UL53 and UL50
S216E/S19E (EE) 53-F AD169-RV Sharma et al. (8) Double mutant carrying glutamate replacements of UL50 serine-216 and UL53 serine-19 in UL53-FLAG genetic background
K355Q 53-F 53-F AD169-RV Sharma et al. (8) UL97 kinase active site mutation (mutation of lysine 355 to glutamine) in UL53-FLAG AD169rv genetic background