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. 2014 Dec 13;12:357. doi: 10.1186/s12967-014-0357-0

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© Chua et al.; licensee BioMed Central. 2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Proposed mechanisms underlying the genetic deletion of DPP4 or therapeutic effect of sitagliptin on protecting heart from acute ischemia-reperfusion injury. IR = ischemia-reperfusion; GLP-1 = glycogen-like peptide-1; DDP4 = dipeptidyl peptidase 4; HO-1 = heme oxygense-1; NQO 1 = NAD(P)H quinone oxidoreductase 1; c-PARP = cleaved poly (ADP-ribose) polymerase; c-Csp 3 = caspase 3; Cyto-Cyt C = cytosolic cytochrome C; TNF-α = tumor necrosis factor alpha; MMP-9 = matrix metalloproteinase 9; NF-κB = nuclear factor κB; ICAM-1 = intercellular adhesion molecule 1; SDF-1 = stromal cell-derived factor 1; LVEDd = left ventricular end diastolic dimension; LVESd = left ventricular systolic dimension; LVEF = left ventricular ejection fraction.