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. 2014 Dec 11;18(6):682. doi: 10.1186/s13054-014-0682-y

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© Granfeldt et al.; licensee BioMed Central. 2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Cardiac function. During infusion of adenosine, lidocaine and magnesium (ALM), the end systolic pressure was significantly lower resulting in an improved arterial–ventricular coupling. Furthermore, systolic and diastolic function evaluated by dP/dt _max and dP/dt _min respectively was improved at the end of the study in the ALM group. (A) End systolic pressure. (B) Maximum rate of pressure development over time (dP/dt _max). (C) Maximum rate of pressure decrease over time (dP/dt _min). (D) Ventricular–arterial coupling. *Significant difference at the end of the study. #Significant different change over time between groups. †Significant different mean/median level during infusion of ALM. ¥Significant difference before/after cessation of ALM infusion. Data presented as median (95% confidence interval). Ea, arterial elastance; Ees, end systolic elastance (end systolic pressure–volume relationship); LPS, lipopolysaccharide.