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. 2014 Nov 15;141(22):4395–4405. doi: 10.1242/dev.110437

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© 2014. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 3. — Mutations with increased growth cone protrusion cause ectopic axon branching. (A) Quantification of VD/DD axon pathfinding defects (guidance and premature termination) and ectopic axon branching in young adults. At least 100 axons were scored. Error bars represent 2× standard error of proportion. (B-D) Representative fluorescence micrographs of VD/DD axons demonstrating axon pathfinding and branching errors. (B) Wild-type, (C) mig-2(mu28); ced-10(n1993M+) and (D) unc-44(e362) VD/DD axons. Arrows point to ectopic axon branches. Dorsal is up, and anterior is left. M+ indicates that the animals had wild-type maternal ced-10(+) activity. Scale bar: 10 µm.