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. 2014 Jun 23;3(3):e000961. doi: 10.1161/JAHA.114.000961

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© 2014 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley Blackwell.

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Figure 8. — AID‐TAT peptide decreases infarct size and supports contractility in vivo. A, Representative sections from rat hearts treated with 1 μmol/L of scrambled (AID(S)‐TAT) or active (AID‐TAT) peptides. Infarct size is assessed as area that did not take up nitroblue tetrazolium dye. B, Mean±SEM of infarct size in hearts following myocardial infarction expressed as a percentage (%) of total left ventricular (LV) area for all hearts treated with 1 or 10 μmol/L of AID(S)‐TAT or AID‐TAT peptide as indicated. C, Mean±SEM of heart/body weight ratio (Hwt/Bwt) for all hearts treated with 10 μmol/L of AID(S)‐TAT or AID‐TAT peptide as indicated. D, Mean±SEM of left ventricular systolic pressure (LVSP) for all hearts treated with 10 μmol/L of AID(S)‐TAT or AID‐TAT peptide as indicated. E, Mean±SEM of maximum dP/dt for all hearts treated with 10 μmol/L of AID(S)‐TAT or AID‐TAT peptide as indicated. F, Mean±SEM of systolic and diastolic arterial blood pressure (BP) for all animals treated with 1 or 10 μmol/L of AID‐TAT peptide as indicated. n represents number of animals. AID indicates alpha‐interacting domain; TAT, transactivator of transcription.