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. 2014 Dec 24;7:94. doi: 10.1186/s13045-014-0094-0

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© Skvarova Kramarzova et al.; licensee BioMed Central. 2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Differing HOX gene expression levels observed in FAB subtypes with respect to the presence of typical molecular aberrations. A. CBFb-MYH11+/− patients with AML M4. Asterisks indicated statistical significance (* ≤ 0.05, ** ≤ 0.01; additional borderline significance: p = 0.0679 for HOXA7, HOXA10 and HOXB9, p = 0.0732 for HOXA13); B. RUNX1-RUNX1T1+/− patients with AML M2. Asterisks indicated statistical significance (* ≤ 0.05, ** ≤ 0.01; additional borderline significance: p = 0.1745 for HOXA5, p = 0.1745 for HOXA9 and P = 0.1172 for HOXA10).