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. Author manuscript; available in PMC: 2016 Jan 1.
Published in final edited form as: Front Neuroendocrinol. 2014 Aug 4;0:49–71. doi: 10.1016/j.yfrne.2014.07.001

Figure 4.

Figure 4

The induction of flank marking by intracerebroventricular injection of oxytocin (OT) is mediated by the vasopressin V1a receptor (V1aR). A. The amount of flank marking induced by the injection of the three concentrations of the selective V1aR agonist (Ag), [Phe2]OVT, OT or two concentrations of the selective OTR Ag, [Thr4,Gly7]OT, in 1 microliter of vehicle. (* and # indicate significant differences compared to the OT group) B. The amount of flank marking induced by the injection of 90 µM OT combined with vehicle or three concentrations of the selective V1a receptor antagonist (A), d(CH2)5[Tyr(Me)2]AVP (Manning Compound), or three concentrations of the selective OTR A, desGly-NH2-d(CH2)5[D-Tyr2,Thr4]OVT. (* indicates significant difference from the 90µM OT group). C. The amount of flank marking induced by the injection of various concentrations of OT and arginine-vasopressin (AVP) (* indicates significant difference between OT and AVP). From Song, McCann, McNeill, Larkin, Huhman and Albers, unpublished data.