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. Author manuscript; available in PMC: 2016 Mar 1.

Published in final edited form as: J Mol Neurosci. 2014 Aug 26;55(3):644–652. doi: 10.1007/s12031-014-0403-7

Fig.6 — Metabolic phenotype and neurobehavioral performance of POKO mice. (A) There was a correlation of percent of body fat and body weight of the POKO and WT mice at age of 22–25 weeks. POKO mice had higher adiposity than WT mice (p < 0.0001). (B) POKO mice showed reduced circadian amplitude of thermal pain latency, with significant decrease at 4 pm and 4 am. (C) Potential interactions of SF and obesity were determined in 4 groups of mice. SF had a trend toward increased pain threshold (p = 0.07) by 2-way ANOVA. SF increased pain threshold in the WT mice, whereas the POKO mice did not show a significant change.

Fig.6 — Metabolic phenotype and neurobehavioral performance of POKO mice. (A) There was a correlation of percent of body fat and body weight of the POKO and WT mice at age of 22–25 weeks. POKO mice had higher adiposity than WT mice (p < 0.0001). (B) POKO mice showed reduced circadian amplitude of thermal pain latency, with significant decrease at 4 pm and 4 am. (C) Potential interactions of SF and obesity were determined in 4 groups of mice. SF had a trend toward increased pain threshold (p = 0.07) by 2-way ANOVA. SF increased pain threshold in the WT mice, whereas the POKO mice did not show a significant change.