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. 2015 Jan 9;17(1):2. doi: 10.1186/s13075-014-0515-z

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© Turunen et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Overview of the antibody specifities in controls (panels A and C) and RA patients (panels B and D) and RA patients’ CCP and MCV results. In panels A and B, antibodies specific to CitI are depicted in dark columns/blue and those specific for HcitI in light columns/green. In panels C and D, antibodies to CitII are presented in dark columns/blue and those to HcitII in light column/green. The values shown are percent of inhibition by respective soluble peptides to binding to the similar immobilized peptide. Panel E shows the binding of RA patient sera to the antigens of the CCP and MCV assays, shown as arbitrary units defined by each manufacturer. CCP, cyclic citrullinated protein; Cit, citrulline; Hcit, homocitrulline; MCV, mutated citrullinated vimentin; RA, rheumatoid arthritis.