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. Author manuscript; available in PMC: 2015 Feb 11.
Published in final edited form as: Am J Kidney Dis. 2011 Nov 9;59(1):152–159. doi: 10.1053/j.ajkd.2011.08.035

Table 2.

Etiologies of hypophosphatemia

Acute hypophosphatemia Chronic hypophosphatemia
Decreased phosphate intake Inadequate parenteral nutrition
Nutritional defects
Decreased intestinal absorption Vitamin D deficiency
Vitamin D-dependent rickets
Chronic anti-acid therapy
Intestinal malabsorption
Chronic liver disease
Alcoholism
Increased renal wasting of primary renal disease After renal transplantation (first weeks)
Acute volume expansion
De De Toni Debre Fanconi syndrome
Dent disease
Toxic (ifosfamide, platin salts, diuretics, glucocorticoids, retroviral therapies in HIV patients, acetazolamide)
Hyperparathyroidism Primary and secondary hyperparathyroidisms
Increased FGF23 serum levels Hypophosphatemic rickets
Tumor-induced osteomalacia
Fibrous dysplasia
Mac Cune Albright syndrome
Toxic (saccharated ferric oxide)
Redistribution of phosphate between the different compartments After bone marrow transplantation
Acute leukemia and lymphoma
Correction of diabetic ketoacidosis
Refeeding
Acute respiratory alkalosis
Hungry bone syndrome
Treatment with erythropoiesis-stimulating agents in patients with cirrhosis
Miscellaneous Severe sepsis
Extensive burns
Inadequate dialysis
Acute paracetamol overdose
Salicylate poisoning
Hypothermia