Table 2.
Acute hypophosphatemia | Chronic hypophosphatemia | |
---|---|---|
Decreased phosphate intake | Inadequate parenteral nutrition Nutritional defects |
|
Decreased intestinal absorption | Vitamin D deficiency Vitamin D-dependent rickets Chronic anti-acid therapy Intestinal malabsorption Chronic liver disease Alcoholism |
|
Increased renal wasting of primary renal disease | After renal transplantation (first weeks) Acute volume expansion |
De De Toni Debre Fanconi syndrome Dent disease Toxic (ifosfamide, platin salts, diuretics, glucocorticoids, retroviral therapies in HIV patients, acetazolamide) |
Hyperparathyroidism | Primary and secondary hyperparathyroidisms | |
Increased FGF23 serum levels | Hypophosphatemic rickets Tumor-induced osteomalacia Fibrous dysplasia Mac Cune Albright syndrome Toxic (saccharated ferric oxide) |
|
Redistribution of phosphate between the different compartments | After bone marrow transplantation Acute leukemia and lymphoma Correction of diabetic ketoacidosis Refeeding Acute respiratory alkalosis Hungry bone syndrome |
Treatment with erythropoiesis-stimulating agents in patients with cirrhosis |
Miscellaneous | Severe sepsis Extensive burns Inadequate dialysis Acute paracetamol overdose Salicylate poisoning Hypothermia |