Table 6.
Hypertension W2 (N=738) |
Rapid heart rate W2 (N=700) |
High CRP W2 (N=631) |
CVD events W2 (N=736) |
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---|---|---|---|---|---|---|---|---|---|---|---|---|
Model 1 | Model 2 | Model 1 | Model 2 | Model 1 | Model 2 | Model 1 | Model 2 | |||||
Controlled vs. normal |
Undiagnosed vs. normal |
Uncontrolled vs. normal |
Controlled vs. normal |
Undiagnosed vs. normal |
Uncontrolled vs. normal |
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Age W1 (ref=57–64) | ||||||||||||
65–74 | 0.45 (0.46) | 0.77 (0.41) | 0.15 (0.37) | 0.43 (0.45) | 0.74 (0.41) | 0.10 (0.37) | −0.13b (0.34) | −0.13 (0.34) | −0.51 (0.27) | −0.57* (0.26) | 0.82* (0.33) | 0.83* (0.33) |
75–85 | −0.88 (0.60) | 0.19 (0.68) | −1.14*b (0.53) | −0.98 (0.60) | 0.09a (0.71) | −1.23*a (0.55) | 0.44 (0.49) | 0.22b (0.55) | 0.03 (0.42) | 0.04 (0.41) | 1.15 (0.62) | 1.27* (0.60) |
NMQ W1 | −0.16 (0.19) | 0.03 (0.20) | −0.18b (0.21) | −0.36 (0.29) | 0.17 (0.30) | −0.24 (0.34) | 0.01 (0.15) | −0.03 (0.19) | 0.33*a (0.13) | 0.22 (0.15) | −0.02 (0.21) | 0.04 (0.32) |
NMQ W1 X age 65–74 | 0.24 (0.51) | −0.35 (0.46) | 0.02 (0.53) | −0.15a (0.43) | 0.38 (0.30) | 0.05 (0.38) | ||||||
NMQ W1 X age 75–85 | 0.61b (0.75) | −0.31a (0.61) | 0.28b (0.74) | 0.62 (0.51) | −0.03 (0.36) | −0.45a (0.52) | ||||||
Change of NMQ W2-W1 | −0.57*a (0.25) | −0.33a (0.20) | −0.16a (0.21) | −1.07**a (0.31) | −0.26 (0.27) | −0.49a (0.25) | ||||||
Change of NMQ W2-W1 X age 65–74 | 1.30*a (0.51) | 0.16 (0.49) | 0.99* (0.45) | |||||||||
Change of NMQ W2-W1 X age 75–85 | 0.66 (0.68) | −0.44 (0.68) | 0.45 (0.71) | |||||||||
Constant | −0.46 (1.11) | 2.92 (1.47) | 1.86* (0.92) | −0.59 (1.07) | 2.96* (1.45) | 1.76 (0.91) | −2.20 (1.40) | −2.26 (1.38) | −1.35 (0.97) | −1.33 (0.96) | −2.23 (1.51) | −2.27 (1.52) |
p<0.001,
p<0.01,
p<0.05.
Wald tests: difference between men and women is significant at p<0.05.
Wald tests: difference between men and women is significant at p<0.1.
Standard errors in parentheses.
NMQ: negative marital quality. W1: Wave 1. W2: Wave 2.
In all models, we control for race-ethnicity, education, relative family income, smoking, drinking, BMI physical activity, antihypertensive medication use, and psychological distress (all measured at Wave 1) as well probability of death at W2 and probability of remaining married in both waves. Wave 1 measures of hypertension, rapid heart rate, high CRP and CVD events are also controlled in all models.
Our preliminary analyses suggested that the effects of change of negative marital quality between W1–W2 were not statistically significant (p > 0.05) when predicting rapid heart rate, high CRP and CVD events for men, and are thus not included in the final models.