Table 1. List of antiviral compounds and concentrations used.

High concentration was defined as the highest non-toxic dose tested based on dose/viability studies in Figure 1. Low concentration represents the value two dilution steps below respective high concentration. A possible mechanism of action for each compound is included; additional mechanisms may also exist.

Compound	Concentrations	Mechanism
Adenosine	1mM & 100μM	Inhibition of pyrimidine synthesis (Schnitzlein and Reichmann, 1980; Stollar and Malinoski, 1981)
Amantadine	1mM & 100μM	Inhibition of endocytosis (Schlegel et al., 1982; Superti et al., 1985)
Ara-A	1mM & 100μM	Inhibition of RNA synthesis (Grant and Sabina, 1972)
Aspirin	3mM & 300μM	Promotion of NO synthesis (Chen et al., 2000)
Chloroquine	30μM & 3μM	Inhibition of endosomal escape (Coombs et al., 1981; Dille and Johnson, 1982; Pérez and Carrasco, 1994)
Dansylcadaverine	100μM & 10μM	Inhibition of endocytosis (Schlegel et al., 1982)
Interferon	100U/mL & 10U/mL	Induction of antiviral genes (Detje et al., 2009; Wollmann et al., 2007)
Mycophenolic acid	300μM & 30μM	Depletion of GTP (Ye et al., 2012)
Octyl-gallate	100μM & 10μM	Unknown. Inhibits viral replication. (Uozaki et al., 2007; Yamasaki et al., 2007)
Ribavirin	3mM & 300μM	Pleiotropic effects. Guanosine analog. (Toltzis and Huang, 1986; Hong and Cameron, 2002; Shah et al., 2010)
Rimantadine	300μM & 30μM	Inhibition of endocytosis (Kolocouris et al., 1996; Mato et al., 1983; Schlegel et al., 1982)
SNAP	1mM & 100μM	Organic NO donor (Bi and Reiss, 1995)

^*Abbreviations: Ara-A, adenine 9-β-D-arabinofuranoside; SNAP S-Nitroso-N-acetylpenicillamine; NO, Nitric Oxide.