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. Author manuscript; available in PMC: 2016 Apr 15.
Published in final edited form as: J Comp Neurol. 2015 Jan 30;523(6):921–942. doi: 10.1002/cne.23724

Figure 9. SpVm is necessary for vibrissa reflex circuit activity.

Figure 9

A: Experimental ketamine-anesthetized setup. A bipolar electrode is positioned for mystacial pad stimulation to displace vibrissae, and activity from the transected buccal branch of the facial nerve is recorded with a suction electrode. Electrolytic lesion is performed in SpVm. B: VIIn multiunit activity in response to electrical stimulation of mystacial pad (mean ± 0.95 C.I.). Electrical activity is filtered and rectified (see Methods). VIIn has two peaks in activity following sensory stimulation, both of which are eliminated following lesion of SpVm. C: Representative histological slices confirming the lesion locations. The total current density and number of lesion locations was constant across experiments.