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. 2014 Jun 17;19(1):307–314. doi: 10.1007/s11325-014-1020-y

Table 4.

Correlation between alcohol-induced blood gas changes in sleep and study population characteristics

N = 26 Change in mean SpO2 a Change in mean ΔPtcCO2
Pearson’s r P value Pearson’s r P value
Demographic data
 Age, years −0.11 0.616 0.02 0.914
 Gender 0.08 0.709 0.06 0.784
 BMI kg/m2 −0.13 0.543 0.13 0.516
Laboratory data
 PaO2, kPa 0.51 0.009 −0.25 0.227
 PaCO2, kPa −0.58 0.002 0.11 0.604
 pH 0.37 0.071 0.05 0.816
Spirometry
 FVC % of pred 0.28 0.170 −0.04 0.855
 FEV1 % of pred 0.32 0.125 −0.03 0.886
 DLCO mmol/min/kPab 0.32 0.161 −0.23 0.308
 RV/TLC ratioc −0.28 0.202 0.01 0.950
Sleep
 Change in REM % of TST 0.12 0.554 −0.15 0.454
 Change in awakenings −0.11 0.602 0.22 0.274
 Change in mean SpO2 −0.30 0.135

Change in mean S p O 2 difference between alcohol and control sleep in the mean oxygen saturation, Change in mean ΔP tc CO 2 difference between alcohol and control sleep in the mean increase from awake, supine, transcutaneous carbon dioxide pressure, BMI body mass index, P a O 2 arterial pressure of oxygen, P a CO 2 arterial pressure of carbon dioxide, FVC % of pred forced vital capacity as percent of predicted value, FEV1% of pred forced expiratory volume first second as percent of predicted value, DLCO diffusing capacity of the lung for carbon monoxide, RV residual volume, TLC total lung capacity, Change in REM % of TST difference in the mean rapid eye movement sleep percent of total sleep time between alcohol and control sleep, Change in awakenings difference in the number of awakenings between alcohol and control sleep

aPartial correlation controlling for LTOT use is tabulated

bFour missing from DLCO because of insufficient vital capacity or because they could not hold their breath for 10 s

cTwo missing from body plethysmography because of claustrophobia