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. 2014 Dec 23;2(12):e12195. doi: 10.14814/phy2.12195

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© 2014 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Figure 6. — Glut4 mRNA and cytosol glut4 protein in C2C12 cells. The cells were untreated (control, C) or treated with ethanol (E), E plus tauroursodeoxycholic acid sodium (T), histone deacetylase (HDAC) inhibitor SAHA (SA), HDAC activator resveratrol (Re), p300 histone acetylase (HAT) activator CTPB (CT), or HAT inhibitor garcinol (Ga). HAT was also inhibited with p300 siRNA (si). p300c was a control solution for siRNA. Data shown as means ± SEM of 3 independent experiments carried out in triplicates. Means with different letters are significantly different (Tukey's test, P < 0.05).