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. 2014 Dec 24;2(12):e12251. doi: 10.14814/phy2.12251

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© 2014 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Figure 8. — Role of CBS and CSE mediated H₂S and glutathione production in inflammatory response in kidney ischemia–reperfusion injury. The role of the CBS/CSE system in proinflammatory cytokine expression is summarized in the context of kidney ischemia–reperfusion (I/R)‐induced acute kidney injury (AKI).