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. Author manuscript; available in PMC: 2016 Jan 1.
Published in final edited form as: Arterioscler Thromb Vasc Biol. 2014 Nov 13;35(1):155–162. doi: 10.1161/ATVBAHA.114.304683

Figure 1. LRP1 protein was uniformly abundant in arterial vasculature, and its absence in SMCs did not lead to aortic pathologies in young mice.

Figure 1

A. Representative immunoblot of LRP1 protein abundance in ascending (Asc), thoracic (Thor), suprarenal (Supra), infrarenal (Infr) aorta, and superior mesenteric artery (SMA). β-actin was used as a loading control. Bar graph depicts LRP1/β-actin ratio quantified for each region. Histobars represent group means and errors are SEMs (n=4 per group). B. Representative immunoblot of vascular SMCs harvested from thoracic and abdominal aortas of smLRP1+/+ and -/- mice. β-actin was used as a loading control. C,D. Maximum suprarenal and ascending aortic diameter measured by ultrasound in 8 week old mice (n= 24 per group).