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. 2014 Dec 24;43(2):775–786. doi: 10.1093/nar/gku1346

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© The Author(s) 2014. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 6. — Bivalenthistone modifications form on H3.3-containing SKM gene loci in mouse embryo. (A) H3.3-enriched regions showed more bivalent modifications than those exhibiting no modifications and tended to colocalize with H3K4me3 and H3K27me3 modifications. (B) The quantitatively balanced state of the bivalent modification was associated with H3.3 incorporation. The scatter plot represents the relative ratio of H3K4me3 and H3K27me3 modifications (x-axis) and H3.3 ChIP-seq signal intensities (y-axis) in tissue-specific genes at TSS ± 2 kb. Related data are shown in Supplementary Tables S6-8.