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. Author manuscript; available in PMC: 2016 Jan 31.
Published in final edited form as: Circ Arrhythm Electrophysiol. 2014 Dec 9;8(1):165–173. doi: 10.1161/CIRCEP.114.001789

Figure 2.

Figure 2

A: Representative intracellular Ca2+ transients recorded at a slow pacing cycle length (300 ms) and normalized peak-to-peak demonstrate a longer duration (measured at 85% of diastole) in MI compared to sham hearts. Summary data shows that Ca2+ transient durations are significantly prolonged in MI hearts (n=6) compared to shams (n=5). B: Western blots against SERCA2a and actin (loading control) for sham and MI hearts. Summary data shows that across all hearts (n=3 for each) SERCA2a protein expression was unchanged in MI.