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. Author manuscript; available in PMC: 2015 Feb 23.

Published in final edited form as: Biochim Biophys Acta. 2010 May 25;1800(9):906–911. doi: 10.1016/j.bbagen.2010.05.002

Fig. 3 — Effects of amiloride and derivatives on the colonic and gastric K-ATPase activity. (A) Colonic K-ATPase activity was measured as a function of the concentration of amiloride. (B) Gastric K-ATPase activity was measured as a function of the concentration of amiloride (□, solid line), or benzamil (○, dashed line), or EIPA (△, dotted line). (C) K-ATPase activity was measured as a function of the concentration of amiloride in the absence (■, solid line) or presence of 1 μM nigericin (●, short dashed line). Values are means ± se of 3 animals.

Fig. 3 — Effects of amiloride and derivatives on the colonic and gastric K-ATPase activity. (A) Colonic K-ATPase activity was measured as a function of the concentration of amiloride. (B) Gastric K-ATPase activity was measured as a function of the concentration of amiloride (□, solid line), or benzamil (○, dashed line), or EIPA (△, dotted line). (C) K-ATPase activity was measured as a function of the concentration of amiloride in the absence (■, solid line) or presence of 1 μM nigericin (●, short dashed line). Values are means ± se of 3 animals.