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. 2014 Dec 16;3(6):e001232. doi: 10.1161/JAHA.114.001232

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© 2014 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley Blackwell.

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Figure 10. — (A) Expression of activating FcγRs in DKO mice. Surface expression of FcγRI (CD64) on PBMCs was evaluated by flow cytometry, using an antibody to CD64 (FcγRI). CD64 levels were not different. (B) FcγRI, III, and IV transcripts were quantified by qRT‐PCR from thioglycolate‐elicited peritoneal macrophages (n=5). Significance was determined by independent sample t test. Although FcγRIII expression trended higher (P=0.06), there were no statistically significant differences in FcγR expression. Apo indicates apolipoprotein; DKO, double knockout; PBMCs, peripheral blood mononuclear cells; qRT‐PCR, quantitative real‐time polymerase chain reaction.