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ADM drives tobacco-induced tumor progression. (A) Dose dependent tumor growth response to systemic injections of increasing concentrations of CSE. (B) Dose dependent gene expression of ADM and AHR responsive genes in xenograft tumors exposed to increasing concentrations of CSE (0.06–0.18 mg tar). (C) Blockage of ADM with the small molecule antagonist NSC16311 inhibits CSE-induced tumor growth. Treatments with NSC16311 started two weeks after injection of tumor cells (red arrow).
