Table 2.
Patient No. | Post-stroke follow-up | Time to follow-up | Topology | Stroke extension |
---|---|---|---|---|
1 | None | |||
2 | CT | 24 h | NC+, Ins+ | MCA M1 occlusiona |
3 | MRI | 24, 120 h | Ins+ | MCA M2 occlusiona |
4 | MRI | 48 h | NC+, Ins+ | MCA M1 occlusiona |
5 | CT | 24 h | NC+, Ins+ | MCA M1 occlusionb |
6 | CT | 24, 120 h | Ins+ | MCA M1 occlusionb |
7 | None | |||
8 | MRI | Chronic | Ins+ | MCA M1 occlusion |
9 | CT | 24, 72 h | NC+, Ins+ | MCA M1 occlusiona |
10 | CT | 24 h | NC+, Ins+ | MCA M1 occlusiona |
Same patient numbering as in Table 1. Two patients received no follow up examination due to early transfer to other hospitals. The remaining eight patients developed permanent tissue damage at the corresponding areas on follow-up imaging. “Chronic” in the column “time to follow-up” denotes cerebral imaging ≥1 year after stroke. The following abbreviations are used: NC+, permanent tissue damage (CT-hypodense, MRI T2/FLAIR hyperintense) in caudate nucleus; Ins+, permanent tissue damage (CT-hypodense, MRI T2/FLAIR hyperintense) in insula; MCA, middle cerebral artery; M1, M1 segment of MCA; M2, M2 segment of MCA
ai.a. Thrombolysis, mechanical recanalisation
bi.v. Thrombolysis