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. 2015 Feb 26;11(2):e1005002. doi: 10.1371/journal.pgen.1005002

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© 2015 Deml et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 7 — Wild-type (A-E) and mutant (F-O) zebrafish embryos at 24–48-hpf were analyzed as indicated in the right bottom corner of each image. Please note a change in pax6b transcript distribution at 24-hpf and 48-hpf in mab21l2 ^Q48Sfs*5 (arrowheads in F-H), retinal folding defect in mab21l2 ^R51_F52del embryos at 24-hpf (arrowheads in K, L) and visibly abnormal pax6b pattern at 48-hpf in some (arrowheads in M) but not all (N) mab21l2 ^R51_F52del embryos. pax2.1 expression seems to be unaffected in 24-hpf frameshift mutant embryos (I) but shows an abnormal pattern in both mutants at 48-hpf (J,O). At 48-hpf, in addition to more broad and intense pax2.1 expression in the region of optic fissure, abnormal pax2.1 staining was detected in central retina in mab21l2 ^Q48Sfs*5 embryos (arrowheads in J). le, lens; of, optic fissure; retina.