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. Author manuscript; available in PMC: 2015 Apr 1.
Published in final edited form as: J Cell Physiol. 2014 Apr;229(4):512–520. doi: 10.1002/jcp.24472

Figure 1. Loss of retromer activity activates the cellular immune response in Drosophila prior to infections.

Figure 1

(A-C) Third instar larvae of (A) wild type, (B) dvps35 mutants and (C) tubGal4-dvps35RNAi. Scale bar, 1 mm. (D) Total hemocyte count of wild type and tubGal4-dvps35RNAi third instar larvae. (E) Lamellocyte ratio in total hemocytes of wild type and tubGal4-dvps35RNAi third instar larvae. (F and G) Lymph glands of (F) tub-Gal4 and (G) tub-Gal4-dvps35RNAi third instar larvae. Anterior, left. Scale bar, 200 μm. (H) phospho-Histone H3 and DAPI staining in lymph glands of tub-Gal4 and tub-dvps35RNAi third instar larvae. (I) Antp and DAPI staining in lymph glands of tub-Gal4 and tubGal4-dvps35RNAi third instar larvae. (J) Cut, DE-cadherin and DAPI staining in lymph glands of tub-Gal4 and tubGal4-dvps35RNAi third instar larvae. Anterior to the top in all images in H-J. Scale bar in H-J, 50 μm.