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. 2012 Feb 15;7(5):347–351. doi: 10.3969/j.issn.1673-5374.2012.05.004

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Copyright: © Neural Regeneration Research

This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Tyrosine kinase receptor (Trk) influence on brain-derived neurotrophic factor (BDNF) inhibition β-amyloid peptide (Aβ)(25–35)-induced cell apoptosis. PC12 cells were treated with Trk inhibitor K252a.

Western blot analysis shows that the effects of BDNF on Aβ(25–35)-induced cell apoptosis are diminished following K252a treatment.

Data are expressed as mean ± SD. Results are expressed as a ratio of control cells. Three independent experiments were performed in duplicate.

^aP < 0.05, vs. controls. Statistical analyses were performed using one-way analysis of variance, followed by the two-tailed Student's t-test.