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. 2015 Mar 10;6:46. doi: 10.3389/fneur.2015.00046

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Copyright © 2015 Reynolds, Miller-Delaney, Jimenez-Mateos, Sano, McKiernan, Simon and Henshall.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Focal-onset SE causes rapid changes in polycomb transcript levels in the hippocampus of sham-preconditioned and seizure-tolerant mice. Relative polycomb transcript expression, as compared to control and corrected to β-actin, was determined using qRT-PCR. All significant differences are indicated in green (up-regulation) or red (down-regulation), as determined versus control following one-way analysis of variance with Tukey post hoc test. Gray cells indicate no significant difference versus control samples and black cells indicate that no data are available for that particular cell. Polycomb members/complexes are indicated and tabulated by timepoint (1, 4, 8, and 24 h), experimental group (epileptic injury and tolerance), and hippocampal subfield (CA3, CA1, and DG). CA, cornu ammonis; DG, dentate gyrus; INJ, injury; KA, kainic acid; PRC, polycomb repressive complex; TOL, tolerance.