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. 2015 Mar 10;10(3):e0118886. doi: 10.1371/journal.pone.0118886

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© 2015 Ioannou et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 1 — Panels A-F provides representative examples of karyotypes and different chromosomal aberrations observed after genotoxic exposure. Panels A-C depict individual metaphase spreads. Panels D-F provides examples of individual chromosome pairs with specific chromosome aberrations (magnified). A description of each panel follows: (A) Normal 46, XX metaphase spread; (B) Metaphase spread displaying distorted pulverized chromosomes with altered morphology, indistinct banding pattern and indistinguishable centromeres (several examples are indicated by arrows); (C) Metaphase spread demonstrating premature sister-chromatid separation (e.g. black arrow) and premature centromere separation (e.g. gray arrow); (D) Gap in chromosome 6; (E) Dicentric chromosome 3 at band 3p21; and (F) Chromatid break in the X chromosome at band Xq21.