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. 2015 Mar 1;15:19. doi: 10.1186/s12883-015-0272-x

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© Long et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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OCR deficits in non-synaptic brain mitochondria from mutant APP expressing mice following NMN treatment. Average OCR (pMoles/min) in non-synaptic mitochondria isolated from APP_(swe)/PS1_(ΔE9) (AD-Tg) and non-transgenic (NTG) mouse (3 months) brain. Solid black bar (NTG vehicle control), white bar (AD-Tg vehicle control) and striped bar (AD-Tg NMN). Data are presented as the average OCR ± SE. N = 3–5 separate animals per group. *p < 0.01.