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. 2015 Mar 13;10(3):e0120044. doi: 10.1371/journal.pone.0120044

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© 2015 Schwartz et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 7 — Target gene expression was normalized to the stable housekeeping gene beta 2 microglobulin (B2M), and further normalized to plantaris tendons that were not subjected to synergist ablation. Values are mean±SD, N = 8 tendons for each group. Differences between groups were tested using a two-way ANOVA (α = 0.05) followed by Newman-Keuls post hoc sorting: a, different (P<0.05) from 3D vehicle; b, different (P<0.05) from 3D p38 MAPK inhibitor; c, different (P<0.05) from 7D vehicle; d, different (P<0.05) from 7D p38 MAPK inhibitor; e, different (P<0.05) from 28D vehicle.