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Astrocytic molecular players in absence epilepsy. GABAergic synapses in the sensory thalamus show a decreased function of the astrocytic GABA transporter GAT-1, while in the somatosensory cortex, interleukin-1β levels are increased. Astrocytic thalamic gap junctions, that is, connexin 30 and connexin 43, also contribute to absence seizure generation. Modified and reproduced with permission from Crunelli and Carmignoto (2013).
